Wiebke Hartmann

A novel and divergent role of granzyme A and B in resistance to helminth infection.

Strongyloides ratti infection modulates B and T cell responses to third party antigens.

Pathogenic nematodes suppress humoral responses to third-party antigens in vivo by IL-10-mediated interference with Th cell function.

Nematode-induced interference with the anti-Plasmodium CD8+ T-cell response can be overcome by optimizing antigen administration.

T-cell-derived, but not B-cell-derived, IL-10 suppresses antigen-specific T-cell responses in Litomosoides sigmodontis-infected mice.

Nematode-derived proteins suppress proliferation and cytokine production of antigen-specific T cells via induction of cell death.

Foxp3⁺ regulatory T cells delay expulsion of intestinal nematodes by suppression of IL-9-driven mast cell activation in BALB/c but not in C57BL/6 mice.

Nematode-induced interference with vaccination efficacy targets follicular T helper cell induction and is preserved after termination of infection.

Cutting Edge: the BTLA-HVEM regulatory pathway interferes with protective immunity to intestinal Helminth infection.

Litomosoides sigmodontis induces TGF-β receptor responsive, IL-10-producing T cells that suppress bystander T-cell proliferation in mice.

Filariae-Retrovirus Co-infection in Mice is Associated with Suppressed Virus-Specific IgG Immune Response and Higher Viral Loads.

Interleukin-9 promotes early mast cell-mediated expulsion of Strongyloides ratti but is dispensable for generation of protective memory.

Rbpj expression in regulatory T cells is critical for restraining T2 responses.

Basophils Are Dispensable for the Control of a Filarial Infection.

Helminth Infections Suppress the Efficacy of Vaccination against Seasonal Influenza.

Limited role of mast cells during infection with the parasitic nematode Litomosoides sigmodontis.

IL-33 facilitates rapid expulsion of the parasitic nematode Strongyloides ratti from the intestine via ILC2- and IL-9-driven mast cell activation.

Elucidating different pattern of immunoregulation in BALB/c and C57BL/6 mice and their F1 progeny.

Eosinophils and Neutrophils Eliminate Migrating Larvae at the Site of Infection in the Context of Extracellular DNA Trap Formation.

A Combination of Deworming and Prime-Boost Vaccination Regimen Restores Efficacy of Vaccination Against Influenza in Helminth-Infected Mice.

Pre-existing helminth infection impairs the efficacy of adjuvanted influenza vaccination in mice.

Corrigendum: Eosinophils and Neutrophils Eliminate Migrating Larvae at the Site of Infection in the Context of Extracellular DNA Trap Formation.

Filarial infections compromise influenza vaccination efficacy: Lessons from the mouse.

Retinoic acid drives intestine-specific adaptation of effector ILC2s originating from distant sites.

The C-type lectin receptor MINCLE interferes with eosinophil function and protective intestinal immunity in Strongyloides ratti-infected mice.