David H. Eidelman

Airway surface liquid: end of the controversy?

T-cell proliferation induced by local application of LPS in the nasal mucosa of nonatopic children.

Involvement of the cysteinyl-leukotrienes in allergen-induced airway eosinophilia and hyperresponsiveness in the mouse.

Chlorine-induced injury to the airways in mice.

Inhibition of allergic airways inflammation and airway hyperresponsiveness in mice by dexamethasone: role of eosinophils, IL-5, eotaxin, and IL-13.

Nitric oxide and collagen expression in allergic upper-airway disease.

Harvesting airway surface liquid: a comparison of two techniques.

Role of toll-like receptor 4 in protection by bacterial lipopolysaccharide in the nasal mucosa of atopic children but not adults.

Protein nitration in chronic sinusitis and nasal polyposis: role of eosinophils.

NOS 1 is required for allergen-induced expression of NOS 2 in mice.

Sarcolemmal damage in dystrophin deficiency is modulated by synergistic interactions between mechanical and oxidative/nitrosative stresses.

Nitration of respiratory epithelial cells by myeloperoxidase depends on extracellular nitrite.

TH17 cytokines induce human airway smooth muscle cell migration.

CD8 positive T cells express IL-17 in patients with chronic obstructive pulmonary disease.

Genetic and histologic evidence for autophagy in asthma pathogenesis.

Cigarette smoke increases TLR4 and TLR9 expression and induces cytokine production from CD8(+) T cells in chronic obstructive pulmonary disease.

Autocrine-regulated airway smooth muscle cell migration is dependent on IL-17-induced growth-related oncogenes.

Th17-associated cytokines promote human airway smooth muscle cell proliferation.

IL-8 production in response to cigarette smoke is decreased in epithelial cells from COPD patients.

Differential roles of CXCL2 and CXCL3 and their receptors in regulating normal and asthmatic airway smooth muscle cell migration.

Genetic deletion of IL-17A reduces cigarette smoke-induced inflammation and alveolar type II cell apoptosis.

Aryl hydrocarbon receptor (AhR) attenuation of subchronic cigarette smoke-induced pulmonary neutrophilia is associated with retention of nuclear RelB and suppression of intercellular adhesion molecule-1 (ICAM-1).

CXCL1 inhibits airway smooth muscle cell migration through the decoy receptor Duffy antigen receptor for chemokines.

Upregulation of IL-17A/F from human lung tissue explants with cigarette smoke exposure: implications for COPD.

Decreased expression of the NF-κB family member RelB in lung fibroblasts from Smokers with and without COPD potentiates cigarette smoke-induced COX-2 expression.

Fibroblast-epithelial cell interactions drive epithelial-mesenchymal transition differently in cells from normal and COPD patients.

The Aryl Hydrocarbon Receptor and the Maintenance of Lung Health.

Neutrophils from severe asthmatic patients induce epithelial to mesenchymal transition in healthy bronchial epithelial cells.

Differential Regulation of the Asthmatic Phenotype by the Aryl Hydrocarbon Receptor.

Involvement of the ACE2/Ang-(1-7)/MasR Axis in Pulmonary Fibrosis: Implications for COVID-19.

Role of Human Antigen R (HuR) in the Regulation of Pulmonary ACE2 Expression.

Improved Detection of Chronic Obstructive Pulmonary Disease at Chest CT Using the Mean Curvature of Isophotes.

Standardized Cannabis Smoke Extract Induces Inflammation in Human Lung Fibroblasts.

Receptor-mediated effects of Δ -tetrahydrocannabinol and cannabidiol on the inflammatory response of alveolar macrophages.

Differential inflammatory profile in the lungs of mice exposed to cannabis smoke with varying THC:CBD ratio.

Dynamic relationship between the aryl hydrocarbon receptor and long noncoding RNA balances cellular and toxicological responses.

Regulation of long non-coding RNA expression by aryl hydrocarbon receptor activation.

Cannabis vaping elicits transcriptomic and metabolomic changes involved in inflammatory, oxidative stress, and cancer pathways in human bronchial epithelial cells.

The effect of cannabis-derived terpenes on alveolar macrophage function.